ABSTRACT
Reperfusion injury is thought to occur during coronary recanalisation but rarely produces clinically significant effects other than arrhythmia. We report an unusual case of Ventricular Septal Defect [VSD] developing after successful disobliteration of the right coronary artery. In this case clinical, electrocardiographic and biochemical evidence of myocardial injury developed 6 hours after successful percutaneous recanalization of the infarct related artery. A rapidly developing VSD soon became apparent necessitating surgical intervention to repair the defect. Unfortunately the patient died soon after surgery
Subject(s)
Humans , Male , Heart Septal Defects, Ventricular/etiology , Myocardial Infarction , Coronary Artery Disease , Stents/adverse effects , ElectrocardiographyABSTRACT
It is an unusual case of bilateral tension pneumothoraces developing in the postoperative period in a patient who underwent total arterial revascularization for two vessel coronary artery disease. The patient had been a previous heavy smoker and at operation had been noted to have thin walled lung parenchyma with multiple small bullae mainly in the left upper lobe. He suddenly developed bilateral pneumothoraces following intermittent continuous positive airway pressure requiring initially bilateral needle decompression followed by chest drain insertion. He recovered well and the air leak sealed after 3 days